Also, the Mail online was perhaps the first (UK paper) posting this yesterday: http://www.dailymail.co.uk/health/a...ebilitating-illness-telltale-signs-blood.html
Today the Telegraph chimed in (with what looks, in part, almost plagerised from the Times): http://www.telegraph.co.uk/news/201...ature-of-chronic-fatigue-syndrome-in-blood-w/
And Me Ascossiation (UK) seem to have duplicated The Times article (possibly in full?), for those (like me) without access to the paper's paywall: http://www.meassociation.org.uk/201...trying-to-hibernate-the-times-30-august-2016/
Yeah, 'hibernation' seems to have been hit upon as a sparky aspect to relate to readerships, possibly making for plenty of misunderstanding that it's talking about a behavioral trait, or bizarrely inappropriate evolutionary throw-back. But of course, in the original instance, Naviaux is talking about a similarity between the cellular
states as seen by looking at their metabolite levels. And that only being one potential source of metabolic stress that would trigger a cell to (independently) trigger a 'dauer' like state. This kind of low-power-mode being useful to avoid replicating local viruses and avoiding other pitfalls.
So it's an important, protective, cellular mechanism, but in CFS it's widespread and just doesn't ever switch off (when it should have). Also, the observation of, on average, 75% of metabolic abnormalities unique to individual patients go beyond characterizing the dauer state, I think, and would surely go a long way to illustrating the basis of the great variety of patient symptoms (and how to address them). One also assumes that the hypometabilic state will amplify any existing problem areas of one's biology.